Why The Foot Pain Is Connected To The Neck Pain: Your Movement Patterns Shape Your Body

In the yoga world, if we get pain somewhere in the body, we take it as a call to action and begin to stretch that particular area. This approach is often ineffective, because in the words of prominent physiotherapist Diane Lee “It’s the victims who cry out, not the criminals.” This statement requires a fundamental shift in perspective – just because something is hurting doesn’t mean that it is the source of the problem. Now why is that? Why does the old pain in your right foot eventually shows up as tension in the neck? This happens because of fascia.

Fascia is that cotton candy-like connective tissue, that for hundreds of years had been carefully scraped off by anatomists to expose muscles and bones, and considered irrelevant. In the last couple of decades, however, fascia has been reclaiming it’s role as a vital whole-body communication network.

So what is fascia and why should we, yoga teachers and practitioners, care about it? Fascia is the connective tissue that serves both as a bag that holds muscles , bones, organs, etc, and the packing material in between those structures. It is comprised mostly of collagen fibers. For example, when you look at an individual muscle, you will see that fascia wraps individual muscle fibers, groups of fibers and muscle as a whole, becoming more dense toward the end and forming a tendon, which then seamlessly blends into the fascia that envelops the bone.

“Without it’s [fascia’s] support, the brain would be runny custard, the liver would spread through the abdominal cavity, and we would end up as a puddle at our own feet.” (1)

So the fascial system is an all-pervading physiological network, as important as the circulatory and nervous systems. It is a vast and truly fascinating subject, if you are interested in how the body works. Here we will focus on two qualities of fascia – its continuity and its ability to transmit tension.

As yoga teachers we always concern ourselves with the idea of connection (hence the definition of yoga as “union” or “linking”), yet we often fall into the mechanistic view of the body as a system of levers and pulleys. We tell students that “this pose stretches this muscle”, as if anything in the body works in isolation. In the world of fascia the muscle is linked to the bone, which is linked to the ligament, which is linked to another bone, and then a tendon and another muscle, etc. It’s perfectly fine to study muscle action, characteristics of ligaments, etc. as long as we remember that they are all part of an interconnected fabric within the body and affect each other constantly.

Beyond linking everything to everything, fascia has an important role of communicating mechanical information by the interplay of pulls and pushes. Just like a snag on a sweater can run across the fabric, the tension is transmitted in the same way from one place in the body to another via a fascial net. A human body is a constant interplay of internal and external forces that need to be balanced and distributed. As a result, there are predictable patterns of tension throughout the body that are necessary to keep us upright and allow a wide range of movement. “Strain, tension (good and bad), trauma, and movement tend to be passed though the structure along these fascial lines of transmission.”(1)

To describe those predictable lines of tension, Thomas Myers had adopted the term myofascial meridians (not to be confused with acupuncture meridians – a bit different). A myofascial meridian basically describes a line of tension that runs through a sheet of fascia that connects and envelops several muscles. I can’t help but think of a silly cartoon from my childhood of a cat and a dog pulling on a sausage link.

It is kind of like that. The casing of the sausage link is like fascia, while muscles form the contents. When it’s pulled in the opposite directions, the tension is created that is transmitted throughout the entire length.

Let’s take a look at two “cardinal “ myofascial meridians: Superficial front line and Superficial back line. Just by looking at them it is obvious that SBL and SFL need to balance each other to support the upright position. If the SBL becomes too tight and shortens, you will end up with a “military” posture with some or all posterior (back) muscles shortened and bunched, and the anterior (front) muscles pulled and strained. Or the reverse can be true as well in a “collapsed” posture with a rounded thoracic spine and flattened lumbar curve. The military posture might come with tight hamstrings, but if you only focus on stretching the hamstrings, you won’t resolve the issue. This is where we need to look at the body wholistically (as a whole) and identify the patterns of tension that run throughout

Some patterns of tension are predictable because of body’s organization; others are unique because of the movement patterns, past injuries, etc. Basically, your body responds to the loads that you put on it. For example, one of my former students who spent 30 years driving a folklift in this position, developed his own unique pattern of tension that spiraled around his body and manifested as severe hip and sacrum pain. Just working on his hips wouldn’t be enough, since his hips were the “victims” of this entire unfortunate movement pattern.

In words of Brooke Thomas, “We become the shapes and movements that we make most of the time.”(2) And those patterns do not go away when we go to a yoga class. If a student of mine is used to hiking her right hip up while walking, she will do the same thing while attempting the tree pose. This is where awareness comes in. If we do our yoga practice on autopilot, we reinforce the patterns that we already have. If we pay close attention to what we are doing, we have a chance to overcome those habitual movement patterns. This is one of the reasons we repeat each pose a few times before we hold it – it gives us an opportunity to examine our movement patterns and correct them if necessary (read more about it).

RESOURCES

1. Thomas W. Myers Anatomy Trains: Myofascial Meridians for Manual and Movement Therapists   – In-depth exploration of fascia and myofascial meridians with extensive list of references.

2. Brook Thomas Why fascia matters  – Free, down-to-earth, fun look at fascia and why it matters.

This aeticle originally appeared on sequencewiz.org and was written by Olga Kabel

Muscle Adhesions: Getting in the Way of Your Performance

WHAT ARE ADHESIONS?

Our bodies contain special protein structures called connective tissue, also know as Fascia. This substance connects each part to other parts and the whole, very much like a flexible skeleton. When this tissue is healthy it is smooth and slippery, allowing the muscles, nerves, blood vessels or organs to move freely and function properly. Imagine a piece of scotch tape, the smooth side is healthy fascia; the sticky side is scar tissue or unhealthy fascia. Rub the tape along your skin, both sides, to "feel" what an adhesion is like. The drag that you feel, the "pulling" sensation, is what an adhesion is like. These adhesions attach to muscles, nerves and lymph decreasing their ability to work properly. You really know when you have an adhesion on a nerve; you get many abnormal sensations like numbness, tingling or pain.

DOESN'T STRETCHING GET RID OF ADHESIONS?

Stretching plays a very important role in the treatment and prevention of injuries but it will not break down adhesions. Adhesions, or scar tissue, are much stronger than normal healthy tissue. Muscle groups can often adhere/bind to one another preventing the normal sliding necessary for full mobility. When an individual performs a stretch, the tissue that lengthens is not the adhered tissue but the healthy tissue. This can actually cause more damage to healthy tissue resulting in the increase of adhesions. Stretching correctly is still essential, but it will never release the restrictions that are already present.

HOW WILL EXERCISES AND STRETCHING THAT DID NOT WORK BEFORE, WORK AFTER (ART) TREATMENTS?

Stretching and exercises are only effective after the dysfunction within the soft-tissue structures have been correctly released. Stretching and exercising dysfunctional tissues will only lead to a dysfunctional result. The combination of finding the origin of the problem, (ART) treatments, functional training, stretching and behavioral modifications will result in long lasting results...

HOW DO OVERUSE CONDITIONS OCCUR?

Over-used muscles (and other soft tissues) change in three important ways:

• Acute conditions (pulls, tears, collisions, etc),
• Accumulation of small tears (micro-trauma)
• Not getting enough oxygen (hypoxia).

Each of these factors can cause your body to produce tough, dense scar tissue in the affected area. This scar tissue binds up and ties down tissues that need to move freely. As scar tissue builds up, muscles become shorter and weaker, tension on tendons causes tendonitis, and nerves can become trapped. This can cause reduced range of motion, loss of strength, and pain. If a nerve is trapped, you may also feel tingling, numbness, and weakness.

This article originally appeared on performance-therapy.com

BOOK ONLINE FOR YOUR MASSAGE THERAPY, DRY NEEDLING AND CUPPING MASSAGE NEEDS TO ADDRESS SCAR TISSUE AND MUSCLE ADHESION ISSUES.

If We Can't Stretch Fascia, What Are We Doing?

When Ida Rolf (developer of the profound therapy, Rolfing) began putting her hands and elbows on people’s skin and applying pressure, creating a slow, sustained stretch, she imagined that she was stretching fascial sheets. Generations of manual therapists have followed her thinking, accepting this explanation to account for the changes felt in tissue tension beneath their hands and the sensations experienced by those who receive this type of therapy.

Ideas change over time

Much of manual therapy has grown largely out of anecdotal experience and tradition. Without the means to directly observe or measure what happened inside of the body, explanations for results had to be created from the “outside” and have largely been guesswork. As manual therapy has moved forward, an interest in understanding exactly how touch affects the body has led to a growing interest in research. With research has come the realization that many explanations of the past are not supported by evidence and are sometimes contradicted by evidence. Science-minded manual therapists have learned to adapt to this information, dropping outdated hypotheses and unsupported claims. While some have found it disconcerting to have cherished notions disproved, others have embraced knowledge and have adapted their conceptual models to fit what is known. They may continue to use modalities that have produced desired results but their understanding of how that comes about changes to fit the evidence.

Such a change is happening in the field of “fascial” therapy. 

When Rolf began her groundbreaking work in manual therapy, she devised a hypothesis in an attempt to explain how changes created by her contact came about. However, in recent years, evidence has challenged those explanations. Robert Schleip, Ph.D., was one of the key organizers of the first Fascia Research Congress and is a highly respected researcher. He is credited with discovering minute contractile fibers in fascia, a discovery whose clinical relevance has not yet been demonstrated but still excited many in the world of fascial therapy just the same. In his two-part article, “Fascial Plasticity: a new neurobiological explanation,” published in 2003 in the Journal of Bodywork and Movement Therapies, Schleip points to studies which contradict the notion that we can change the shape of fascia with our hands. One study found that collagen fibers would only begin to stretch shortly before they reached the breaking point, something that would not be desirable in a living human being. In other studies, Schleip, Trager, and others have done Rolfing under anesthesia and found that it produced no results. If the application of manual pressure had the ability to stretch fascia, there should have been a change in spite of anesthesia blocking any neural response. Why, then, was there no change when anesthesia took the nervous system out of the picture?

A neurobiological explanation

If we aren’t stretching fascia, then how do we account for the “release” felt by both the practitioner and the subject? Schleip and others have suggested that the change in tonus is not achieved by an alteration in the shape of fascia but is instead controlled by the nervous system. Schleip suggests that one possible mechanism of change brought about by sustained manual pressure could be the Ruffini corpuscles.

Why Ruffini corpuscles? Clinically, we observe that applying a slow, extended stretch to the skin can create desirable changes both locally and centrally, decreasing tension in the area where the hands are applied as well as creating an overall sense of relaxation. Ruffini corpuscles respond to lateral skin stretch, that is, stretching the skin tangentially or along the same plane as the tissue below. They are slow-adapting, meaning that they continue firing for as long as the stretch is sustained, unlike some mechanoreceptors which respond briefly to new stimulation and then stop responding if it continues.

We know that when we apply our hands to the skin of the body, we stimulate mechanoreceptors. Impulses are sent through the sensory nerves to the brain. The brain evaluates and responds, sending out impulses of its own through nerves to various parts of the body, causing changes to occur in the diameter of blood vessels, breathing, muscle tonus. If it likes our touch, it can create the changes we associate with relaxation, release of tension, and can decrease the sensation of pain. If it feels threatened by our touch, it will do the opposite. As manual therapists, we are always trying to create changes that make the body feel at ease. We can achieve this through the nervous system.

The nervous system is constantly monitoring its environment, responding to a complex array of input. It would be naive and simplistic to think that response to our touch could be reduced to one set of mechanoreceptors or to ignore all the other countless factors. However, when examining the kind of manual therapy we have come to think of as "fascial," understanding the role of Ruffini corpuscles is a good place to start.

Why does it matter?

Does it matter whether we believe we are stretching fascia or not? It matters that we think accurate thoughts about how the body works and what effect our touch has on the body. Understanding how the body actually works will help us work more effectively.

We may still use our hands in ways that we have before. If those methods work to achieve the client's goal, there is no need to abandon them. However, we want to know that how we think about what we are doing is accurate and we want to be able to communicate honestly with our clients. If we discover that our conceptual model is contradicted by what is known about how the body works, then it is time to adapt our model so that our thinking is in agreement with evidence.

Manual therapists need not feel threatened by the news that we cannot stretch fascia. A growing number of Rolfers, practitioners of myofascial release, and related modalities are continuing to use their hands in the ways that have worked for them in the past while adapting their thinking to an updated neurobiological explanation. Many have found that this shift to thinking about the role of the nervous system in manual therapy has led to new, even more effective approaches.
 

A thought experiment

Schleip proposes an interesting thought experiment. During the time it took to read this article, one’s bottom, if seated, is subjected to more pressure over a longer period of time than most therapists will apply to the hips of a client. Yet most of us are not all stretched out and droopy from daily sitting for extended periods of time. Think about it.

This article originally appeared on www.massage-stlouis.com and was written by Alice Sanvito, LMT

The efficiency of the body: Fascia and your health

Tensegrity and the body

Tensegrity is an elision of ‘tension + integrity’. Buckminster Fuller, building on the highly original sculptures of Kenneth Snelson, coined the term, to indicate that the integrity of the structure derived from the balance of tension members, not the compression struts. Can you see? (The easiest way to understand tensegrity is to have a model in your hand – then these properties are self-evident.) Cut the strings and it would collapse totally.

Most of our houses and other man-made structures are ‘compressionegrities’ – their integrity lies with the continuity of compression from the highest brick in the Empire State Building to the lowest block of granite – the compression runs in an unbroken line from element to element all the way to the ground.

Tom and Dr. Steven Levin, developer of biotensegrity

We have thought of our bodies in the same way: the skeleton is a stack of bones, like a stack of checkers – a continuous compression structure – with the individual muscles hanging off each bone to move it.

But every classroom skeleton you have ever seen is wired together. Similarly, in the actual skeleton the bones float in a sea of soft-tissue.

Fascial continuity suggests that the myofascia acts like an adjustable tensegrity around the skeleton – a continuous inward pulling tensional network like the elastics, with the bones acting like the struts in the tensegrity model, pushing out against the restricting ‘rubber bands’.

Tensegrity’s unique features

Playing with these models reveals several unique features: Put strain into a tensegrity structure and the deformation will get distributed all over the structure. Continuous compression structures like buildings do not show this property, but bodies do. Load one corner of a building with a huge amount of snow, and that corner might collapse, leaving the rest of building intact. Load a tensegrity, and it will distribute the strain around the whole structure. Ergo: Where will a strained compression structure break? where the strain is greatest. Where will a tensegrity structure break under strain? at its weakest point.  If we are a tensegrity structure, the ‘load’ that is causing pain or strain in the low back may be sourced in the foot or the shoulder – so we have to be able to see the pattern to know where to intervene.  Erik Dalton says “Don’t chase the Pain!”.  Ida Rolf said :Where you think it is, it ain’t.”  Both of these point to the tesnegrity nature of the body.

Expansion
Often our clients’ bodies are contracted and retracted and immobilized in some ways, in some parts. We want to engender an opening, expansion, a filling of space until the person is fully expressed, not constrained. Tensegrities expand in all axes at once: open the structure in one dimension and every dimension expands. Neither buildings nor balloons display this property, but tensegrity structures and bodies do.

Research into fascia has shown how it works on many levels as a distributive network. The body is at least responding like a tensegrity structure, and many of us believe that it is operating as a tensegrity structure. Perhaps it operates more like a tensegrity structure in Fred Astaire than it does in Jackie Gleason.

Efficiency
Tensegrities can be built in hierarchies – each element of a tensegrity can be built out of smaller tensegrities – making for the most efficient use of materials, an evolutionary imperative. This efficiency is also a very important property, as the rule of biological evolution is efficiency – getting the most performance from the least material.

Research into fascia has shown how it works on many levels as a distributive network. The body is at least responding like a tensegrity structure, and many of us believe that it is operating as a tensegrity structure. Perhaps it operates more like a tensegrity structure in Fred Astaire than it does in Jackie Gleason.

This article originally appeared on www.anatomytrains.com